ARDS or pulmonary oedema?
نویسندگان
چکیده
منابع مشابه
Pulmonary epithelial permeability in ARDS and cardiogenic pulmonary oedema.
Clearance of inhaled 99mTechnetium-labelled diethylene triamine pentacetate (99mTc-DTPA) from the lung, an index of pulmonary alveolar epithelial permeability (PAEP), was measured in 13 patients with cardiogenic interstitial pulmonary oedema (CIPO) and in 7 patients with adult respiratory distress syndrome (ARDS). Thirty-five normal subjects (22 nonsmokers and 13 smokers) were evaluated as cont...
متن کاملVEGF levels in the alveolar compartment do not distinguish between ARDS and hydrostatic pulmonary oedema.
Although overexpression of vascular endothelial growth factor (VEGF) 165 in the lung causes pulmonary oedema, its role in human acute lung injury (ALI) is unclear. VEGF levels are reported to be lower in bronchoalveolar lavage from ALI patients compared with normals, but these studies did not include a comparably ill control group with noninflammatory pulmonary oedema. The current authors hypot...
متن کاملBronchoalveolar and systemic cytokine profiles in patients with ARDS, severe pneumonia and cardiogenic pulmonary oedema.
The aim of this study was to investigate whether bronchoalveolar lavage (BAL) and serum levels of proinflammatory cytokines discriminate between different entities of patients with acute respiratory failure. BAL and circulating concentrations of interleukin-6 (IL-6), interleukin-8 (IL-8) and tumour necrosis factor-alpha (TNF-alpha) were measured in 74 mechanically-ventilated patients and 17 hea...
متن کاملRe-expansion pulmonary oedema.
Clinical details are given of two patients who developed ipsilateral pulmonary oedema following re-expansion of their spontaneous pneumothoraces by intercostal drainage of air. The possible mechanisms underlying the oedema are discussed, and prior literature is analysed. Reference is made particularly to its predictability and to precautions recommended to minimize the frequency of this potenti...
متن کاملPulmonary oedema: physiological considerations.
In this paper I propose to attempt to evaluate our knowledge of the mechanisms of acute pulmonary oedema in experimental animals, and to enquire how far it can be applied to our understanding of the phenomenon in man. Our knowledge of the mechanisms of lung oedema is extremely meagre; yet in some respects it may be integrated, with advantage to future investigations. The first issue which shoul...
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ژورنال
عنوان ژورنال: Anaesthesia
سال: 1996
ISSN: 0003-2409
DOI: 10.1111/j.1365-2044.1996.tb13668.x